Research interest
Academic and Scientific education
Additional Information
selected publications
1. Auger J.-P., Zimmermann M., Faas M., Stifel U., Chambers D., Krischnacoumar B., Taudte V., Grund C., Sholtysek C., Uderhardt S., Maté M.P., Stoll C., Böttcher M., Palumbo-Zerr K., Mangan M.S.J., Dzamukova M., Kieler M., Hofmann M., Blüml,S., Schabbauer G., Mougiakakos D., Sonnewald U., Hartmann F., Simon D., Kleyer A., Grüneboom A., Finotto S., Latz E. Hofmann J., Schett G. Tuckermann J., and Krönke G. (2024). Mitochondrial rewiring essentially contributes to the anti-inflammatory effects of glucocorticoids. Nature 629(8010):184-192. doi: 10.1038/s41586-024-07282-7.
2. Caratti, G., Stifel, U., Caratti, B., Jamil, A.J.M., Chung, K.J., Kiehntopf, M., Graler, M.H., Bluher, M., Rauch, A., and Tuckermann, JP (2023). Glucocorticoid activation of anti-inflammatory macrophages protects against insulin resistance. Nat Commun14, 2271. doi: 10.1038/s41467-023-37831-z
3. Loft A, Schmidt SF#, Caratti G, Stifel U, Havelund J, Sekar R, Kwon Y, Sulaj A, Chow KK, Alfaro AJ, Schwarzmayr T, Rittig N, Svart M, Tsokanos FF, Maida A, Blutke A, Feuchtinger A, Møller N, Blüher M, Nawroth P, Szendrödi J, Færgeman NJ, ZeigererA, Tuckermann J#, Herzig S#. (2022) A macrophage-hepatocyte glucocorticoid receptor axis coordinates fasting ketogenesis. Cell Metab. 34(3):473-486.e9. doi: 10.1016/j.cmet.2022.01.004. # equal correspondence
4. Vettorazzi S, Bode C, Dejager L, Frappart L, Shelest E, Klaßen C, Tasdogan A, Reichardt HM, Libert C, Schneider M, Weih F, Henriette Uhlenhaut N, David JP, Gräler M, Kleiman A, Tuckermann JP. (2015) Glucocorticoids limit acute lung inflammation in concert with inflammatory stimuli by induction of SphK1. Nat Commun. 6:7796. doi: 10.1038/ncomms8796.
5. Rauch A, Seitz S, Baschant U, Schilling AF, Illing A, Stride B, Kirilov M, Mandic V, Takacz A, Schmidt-Ullrich R, Ostermay S, Schinke T, Spanbroek R, Zaiss MM, Angel PE, Lerner UH, David JP, Reichardt HM, Amling M, Schütz G, Tuckermann JP.(2010) Glucocorticoids suppress bone formation by attenuating osteoblast differentiation via the monomeric glucocorticoid receptor. Cell Metab. 11(6):517-31. doi: 10.1016/j.cmet.2010.05.005.
