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Induced global deletion of glucocorticoid receptor impairs fracture healing.
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The glucocorticoid receptor in recipient cells keeps cytokine secretion in acute graft-versus-host disease at bay.
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Deletion of Menin in craniofacial osteogenic cells in mice elicits development of mandibular ossifying fibroma.
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Endogenous glucocorticoid signaling in chondrocytes attenuates joint inflammation and damage.
FASEB journal : official publication of the Federation of American Societies for Experimental Biology, 32 :478-487
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Tayfun;
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Martin;
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Lrp1 in osteoblasts controls osteoclast activity and protects against osteoporosis by limiting PDGF-RANKL signaling.
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Galuppo,
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Bauersachs,
Johann;
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The glucocorticoid receptor in monocyte-derived macrophages is critical for cardiac infarct repair and remodeling.
FASEB journal : official publication of the Federation of American Societies for Experimental Biology, 31 :5122-5132
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The glucocorticoid receptor in monocyte-derived macrophages is critical for cardiac infarct repair and remodeling.
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Bernardo S.;
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Marco;
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Jan;
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Martin;
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Molecular Integration of Incretin and Glucocorticoid Action Reverses Immunometabolic Dysfunction and Obesity.
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Airway Epithelial Cells Are Crucial Targets of Glucocorticoids in a Mouse Model of Allergic Asthma.
Journal of immunology (Baltimore, Md. : 1950), 199 :48-61
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Zhi-Yan;
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Sung-Hyeok;
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Tirode,
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Jan;
Toretsky,
Jeffrey A.;
Kenner,
Lukas;
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Heinrich;
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Sean;
Sweet-Cordero,
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Nakamura,
Takuro;
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Richard;
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Olivier;
Üren,
Aykut
Combined experience of six independent laboratories attempting to create an Ewing sarcoma mouse model.
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Ulf H.;
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Mario M.;
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Mona;
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Rainer;
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Martina;
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Jean-Pierre;
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Philippe;
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Chang X.;
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Jan P.
Loss of menin in osteoblast lineage affects osteocyte-osteoclast crosstalk causing osteoporosis.
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Lea;
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Sabine;
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Katrin;
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Timo D.;
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Matthias H.;
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Christoph;
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Johannes;
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Thomas;
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Natalie;
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Jan P.;
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Adipocyte Glucocorticoid Receptor Deficiency Attenuates Aging- and HFD-Induced Obesity and Impairs the Feeding-Fasting Transition.
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Maja;
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Johannes;
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Piotr;
Jirsa,
Milan;
Deschemin,
Jean-Christophe;
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Sophie;
Trautwein,
Christian;
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Pavel
Hepcidin knockout mice spontaneously develop chronic pancreatitis owing to cytoplasmic iron overload in acinar cells.
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Hepcidin knockout mice spontaneously develop chronic pancreatitis owing to cytoplasmic iron overload in acinar cells.
The Journal of pathology, 241 :104-114
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101.
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H. Herschel;
Henning,
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Activation of dimeric glucocorticoid receptors in osteoclast progenitors potentiates RANKL induced mature osteoclast bone resorbing activity.
Bone, 93 :43-54
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Activation of dimeric glucocorticoid receptors in osteoclast progenitors potentiates RANKL induced mature osteoclast bone resorbing activity.
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Antunica-Noguerol,
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Ana C.;
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GR-independent down-modulation on GM-CSF bone marrow-derived dendritic cells by the selective glucocorticoid receptor modulator Compound A.
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Malehmir,
Mohsen;
Healy,
Marc E.;
Weber,
Achim;
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Mathias;
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Martin;
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Chemical Hybridization of Glucagon and Thyroid Hormone Optimizes Therapeutic Impact for Metabolic Disease.
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Rahil;
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Aryee,
Dave Nt;
Wiedner,
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Tomazou,
Eleni M.;
Logan,
Malcolm;
Hartmann,
Christine;
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Jan P.;
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Lukas;
Mikula,
Mario;
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Helmut;
Üren,
Aykut;
Richter,
Günther H.;
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Increased survival and cell cycle progression pathways are required for EWS/FLI1-induced malignant transformation.
Cell death & disease, 7 :e2419
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Increased survival and cell cycle progression pathways are required for EWS/FLI1-induced malignant transformation.
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97.
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Baumgart,
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Groth,
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Jan P.;
Baschant,
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Dicer ablation in osteoblasts by Runx2 driven cre-loxP recombination affects bone integrity, but not glucocorticoid-induced suppression of bone formation.
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Schmidt,
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Ahmad,
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Lin,
Yu-Chieh;
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Konrad J.;
Tuckermann,
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Ploubidou,
Aspasia
High-Content Microscopy Analysis of Subcellular Structures: Assay Development and Application to Focal Adhesion Quantification.
Current protocols in cytometry, 77 :12.43.1-12.43.44
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High-Content Microscopy Analysis of Subcellular Structures: Assay Development and Application to Focal Adhesion Quantification.
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Jutta;
Weiss,
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Transforming Growth Factor β1 (TGF-β1) Activates Hepcidin mRNA Expression in Hepatocytes.
The Journal of biological chemistry, 291 :13160-74
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Transforming Growth Factor β1 (TGF-β1) Activates Hepcidin mRNA Expression in Hepatocytes.
The Journal of biological chemistry, 291 :13160-74
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